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2000
Volume 23, Issue 8
  • ISSN: 1871-5206
  • E-ISSN: 1875-5992

Abstract

The current study demonstrates amygdalin’s (vitamin B17) postulated mechanism of action on cancer cells where it kills cells by selective toxicity, promotes apoptosis via cell cycle arrest, induces apoptosis via intrinsic cell death pathway (the mitochondria-initiated pathway), and enhances immunity. Thus, amygdalin can be considered a valuable natural cancer therapeutic agent. The toxicity of Amygdalin was reviewed. Moreover, solutions to avoid the cyanide poisoning have been proposed.

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/content/journals/acamc/10.2174/1871520623666221124143751
2023-05-01
2025-03-15
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/content/journals/acamc/10.2174/1871520623666221124143751
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  • Article Type:
    Review Article
Keyword(s): Amygdalin; caspase3; cytochrome c oxidase; mechanism; p53; vitamin B17
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